タイトルまたは説明

GITR (glucocorticoid-induced tumor necrosis factor receptor)
GITR (glucocorticoid-induced tumor necrosis factor receptor), also known as AITR or TNFRSF*8, is a new member of the TNFR superfamily and plays a role in immune regulation.
So far, people have studied more about the role of GITR signal in the acquired immune system, but the research on innate immunity is limited. Innate immunity is an important part of body immunity. Mononuclear macrophages are processing antigens, activating lymphocytes, and phagocytosis Bacteria and other aspects have important functions. When effector T cells are activated, there will be a temporary upregulation of GITR. When the GITR signal is triggered, the inhibitory effect mediated by TGFβ*1 can be abolished.
Human GITR (Histag)                     Human GITR (Fc tag)
Expression host: HEK**3                  Expression host: HEK**3
Molecular weight: *5 kD                   Molecular weight: ****2kD
Concentration: **2mg / ml                  Concentration: **2mg / ml
Purity:> *0%                            Purity:> *0%
Storage environment: 1 × PBS (pH7.4)       Storage environment: 1 × PBS (pH7.4)
Storage conditions: short-term storage at 4℃, long-term storage at **0℃. Avoid repeated freezing and thawing.
Safety Tips: Not for human experiment